bookmark_borderResidential Real Estate Flood Risk Information

Do you know if you need flood insurance? Do you know if you have flood insurance? What happens when your house gets flooded?

Flood risk information has not been readily available to the average consumer. Climate change is making your flood risk drastically increase.

Now, one of the major real estate databases (Realtor.com) is making flood information available to the public.

In the NPR story, Major Real Estate Website Now Shows Flood Risk. Should They All?, experts explain the trend.

“People are buying property with little knowledge of whether it’s going to flood or not,” says Harriet Festing, co-founder of the advocacy group Higher Ground, which connects people across the country who have survived floods. “It ruins lives.”

Millions of buyers risk overpaying for homes likely to be hit with a natural disaster over the course of a 30-year mortgage. But representatives for other realty sites say home sellers are reluctant to publish flood risk information, since it could decrease their home’s value.

What you need to know about flood insurance and your home.

bookmark_borderCOVID-19 Long Haulers: SARS-CoV-2 Causes Dysfunctional Autoimmune Response

Coronavirus / COVID
By Daniel Brouse
Email: help@membrane.com
Last Updated December 18, 2021

Long haulers syndrome is in part due to “an autoimmune response that makes you feel symptoms in waves, or due to stress, and anxiety that triggers your immune system.”
— Daniel Brouse (August, 2020)

My original description of long-COVID was, “It feels like my body is having lots of micro cytokine storms.” Now two years later, the hypothesis has been proven true. “‘It is important to note that the severity of disease in humans is not determined only by virus replication but also by the host immune response to the infection, which may lead to dysregulation of the innate immune system, i.e. “cytokine storm”‘ (Omicron SARS-CoV-2 can infect faster and better)

My gravest concerns about long-COVID remain the same — what are the long term consequences of my immune system attacking my healthy body?

Do recovered coronavirus SARS-CoV-2 patients have immunity?
Why do some people appear to be reinfected?

Maybe. Those who recover from COVID-19 appear to have some immunity.(1) “It’s reasonable to assume that there will be protection for a time frame of one to three years,” says Florian Krammer.(2) Though recent examples suggest immunity, if any, may be very short lived. The Omicron variant appears to escape immunity from both prior infection and vaccination.

It appears there are 2 different types of COVID damage: direct damage from the COVID infection, as well as, a variety of post-COVID syndromes. The direct damage appears to be dependent on the severity of the COVID infection (which likely has to do with viral load, age, and underlying conditions.) The post-COVID long haulers syndrome appears to have little to do with the severity of the COVID infection and more to do with the post-infection immune response.

Then, why do some people appear to get reinfected?
“Over the last 2 weeks, I’ve been working with my doctor about recurring symptoms. It turns out I have ‘long haulers’ syndrome. Every couple of weeks I go through a wave of symptoms. Though not as severe as the original experience, it is still not pleasant. The symptoms don’t come on all at the same time, rather they cycle through over two weeks or so (and not necessarily in the same order.) I’m not infected again, nor am I contagious.(3) The symptoms include:
* difficulty breathing (feeling like you’re not taking in enough oxygen)
* congestion / mild cough
* stomach issues / diarrhea
* loss of taste, smell, and appetite
* night sweats
* nightmares / vivid dreams
* dizziness / the spins / foggy thinking
* headaches / body aches / soreness
* fatigue
* ear aches

What causes the recurring COVID symptoms?
It is too early for a definitive answer; however, an examination of cases appear to point to an autoimmune response due to epigenetic changes caused by the virus.

Is the person still contagious? No.

How long will it take for the immune system to recognize that there is no longer a threat? It’s too early to tell. So far, symptoms have been observed for over 2 years. BUT, there are still many questions about COVID-19 and autoimmune and autoinflammatory diseases.

“Emerging reports show that severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2) infection precedes the appearance of various autoimmune and autoinflammatory diseases, including paediatric inflammatory multisystemic syndrome (PIMS) or multisystem inflammatory syndrome in children (MIS-C), thus adding to the growing mystery of this virus and raising questions about the nature of its link with autoimmune and autoinflammatory sequelae.” (4)

What is long haulers syndrome?
The vast majority of long haulers experience fluctuations both in the type and intensity of symptoms over the course of being symptomatic.(5)

Research is pointing to “an autoimmune response due to changes in the genes of the infected host” for causing “long haulers syndrome” in adults. “Support groups on Facebook include thousands of people who say they have been wrestling with serious COVID-19 symptoms for at least a month, if not two or three. The groups have coined a name for themselves: ‘long-haulers.’ But their lives have been irrevocably changed by the onslaught of symptoms — relentless and rolling waves of fever, headache, nausea, and the terrifying inability to catch their breath. For Roberts, it’s robbed her of time. ‘I was so good. This COVID-19 has stolen my life,’ she says.” (6)

According to a WHO report, most people fall into one of two groups when it comes to the virus. Approximately 80% of those with COVID-19 end up having a mild response and most of those cases resolve in about two weeks. For people who have a severe response to the virus, it can take between three and six weeks to recover.

But now, there is growing concern over a separate group that doesn’t seem to fall into either of those categories. A number of people are now reporting lingering symptoms of the illness for one, two or even three months. This new group is mixed with those who experienced both mild and severe cases. As health experts step in to try to manage these patients and learn more, many are referring to this group as coronavirus “long-haulers” or “long-termers.”

“We’re now seeing a percentage of patients whose symptoms seem to be lasting a while,” explains family medicine provider Christopher Babiuch, MD. “This is challenging because everyone’s needs are so unique. We’re finding that collaborating as a team between different specialists helps to manage and support these patients, but there’s a lot that we just don’t know yet.” (7)

What is actually causing the immune system to have an autoimmune disconnect?
All the research is still theoretical. More studies and longer recovery times are required to confirm the theories. So, we still do not know for sure about the antigen presentation of COVID-19.

Possibly for the longhaulers it’s cytokine imbalance which causes an Down Regulating NAD+, * Up Regulating IDO, Rogue Antibodies, and other conditions that result from genetic changes. The symptomatology improves vastly with NAD+ precursors but if cellular damage or organ damage is to occur the damage is likely permanent.

“The SARS-CoV-2 hyperinflammatory response is associated with high mortality. This hypothesis suggests that a deficiency of nicotinamide adenine dinucleotide (NAD+) may be the primary factor related to the SARS-Cov-2 disease spectrum and the risk for mortality, as subclinical nutritional deficiencies may be unmasked by any significant increase in oxidative stress.

NAD+ levels decline with age and are also reduced in conditions associated with oxidative stress as occurs with hypertension, diabetes and obesity. These groups have also been observed to have high mortality following infection with COVID-19. Further consumption of NAD+ in a pre-existent depleted state is more likely to cause progression to the hyperinflammatory stage of the disease through its limiting effects on the production of SIRT1,” said Ade Wentzel, co-author of the research.(9)

Does an auto-immune disconnect explain all long haulers’ symptoms?
No. Evidence is mounting that long haulers are unique and may have a variety of ailments. Many post-viral COVID cases exhibit the Lyme-like autoimmune disfuntions. Many also exhibit symptoms, such as, dysautonomia (10)(11). Long haulers have reported over 100 different symptoms. COVID appears to impact many of the body’s systems including respiratory, circulatory, cardiovascular, neurological, and immune systems to name a few. This makes diagnosis and treatment very confusing for doctors. And, those aren’t the only difficulties. An additional problem for diagnosing is memory T-cells. Some people’s memory T-cells may be getting erased. That means your body does not recognize previous infections nor vaccines. So, long haulers syndrome may be a combination of new conditions (Lyme-like autoimmune, dysautonomia, chronic fatigue, etc.) AND re-infections to everything you use to be immune to… with no one person having the same ailments as another.

Is there a treatment or cure for long haulers syndrome?
No. Currently, there is no cure or treatment for the onset of these autoimmune responses. The closest comparison appears to be when Lyme disease was first discovered or HIV.

Ade Wentzel replied: You can take nicotinic acid orally or niagen (if you have deep pockets). The IV will help, but however you supplement, you need to keep going for at least a good few months.

Ade has developed a protocol for a NAD+ deficiency: NAD+: Vitamin B3, Vitamin D and sunlight, Vitamin C, Quercetin, Zinc, and Selenium

COVID-19 Index


FOOTNOTES:
0. Technically speaking a virus is never alive. It is only RNA. Dead is not the scientifically correct way to describe it. But, in all essence, the virus is dead.

REFERENCES:
1. Potent antibodies found in people recovered from COVID-19 (NIH)
2. How Long Will Immunity To The Coronavirus Last? (NPR)
3. COVID-19 Testimonial (The Membrane Domain)
4. Autoimmune and inflammatory diseases following COVID-19 (Nature)
5. COVID-19 Prolonged Symptoms Survey – Analysis Report (MIT)
6.What It’s Like When COVID-19 Lasts For Months (NPR)
7. What It Means to Be a Coronavirus “Long-Hauler” (Cleveland Clinic)
8. Study: COVID-19 antibodies decay quickly after mild illness (University of Minnesota Center for Infectious Disease Research and Policy)
9. COVID-19: NAD+ deficiency may predispose the aged, obese and type2 diabetics to mortality through its effect on SIRT1 activity (Science Direct)
10. Dysautonomia: An Overlooked Neurological Manifestation in a Critically ill COVID-19 Patient (NIH)
11. Redefining Covid-19: Months after infection, patients report breathing difficulty, excessive fatigue (CNN)

ADDITIONAL REFERENCES:
A. Long-term consequences of COVID-19: research needs (The Lancet)
B. Humoral Immune Response to SARS-CoV-2 in Iceland (New England Journal of Medicine)
C. Is a Bradykinin Storm Brewing in COVID-19? (The Scientist)
D. The Power of Antibody-Based Surveillance (New England Journal of Medicine)
E. Coronavirus Infection and PARP Expression Dysregulate the NAD Metabolome: A Potentially Actionable Component of Innate Immunity bioRxiv
G. Relationships between hyperinsulinaemia, magnesium, vitamin D, thrombosis and COVID-19: rationale for clinical management British Cardiovascular Society

bookmark_borderCOVID-19 Testimonial

COVID=19
By Daniel Brouse
help@membrane.com

I’ve been reluctant to talk about my COVID experience for several reasons, but have decided — if my testimony can convince just 1 person to avoid contracting or spreading the disease, it will be worth it.

I had COVID back at the end of January and the first weeks of February and conducted a study of people that got infected along with me. The sample size of infected individuals in my study sample is 6.
Ages 30-80
16.7% died
33.3% had severe symptoms
33.3% had moderate symptoms
16.7% were asymptomatic
Age and blood type appear to be the dominant factors.

I’ve been working with my doctor for a couple months. In May, he first told me not to bother with the antibody test. That my symptoms were most certainly COVID. Insurance only covers the antibody test if you have previously tested positive, and getting tested doesn’t tell you anything, anyway. (eg inaccurate test results, inability of tests to determine the amount of antibodies and T cells, etc.) He believes I’ll have immunity until around December.

Now, here’s the interesting part…
I’ve been questioning whether recovered patients have immunity and how some people have appeared to be reinfected? Over the last 2 weeks, I’ve been working with my doctor about recurring symptoms. It turns out I have “long haulers” syndrome. Every couple of weeks I go through a wave of symptoms. Though not as severe as the original experience, it is still not pleasant. The symptoms don’t come on all at the same time, rather they cycle through over two weeks (not necessarily in the same order.) I’m not infected again, nor am I contagious. Exercise or anything that causes inflammation aggravates my symptoms. The symptoms include:

* difficulty breathing (feeling like you’re not taking in enough oxygen)
* congestion / mild cough
* stomach issues / diarrhea
* loss of taste, smell, and appetite
* temperature regulation / night sweats
* nightmares / vivid dreams
* dizziness / the spins / foggy thinking
* headaches / body aches / soreness
* fatigue
* ear aches
* peripheral neuropathy (tingling of the hands and feet)

There is currently no treatment or cure for long haul syndrome.

IMPORTANT UPDATE (November 2020)
A treatment has been found for long haulers syndrome. After 8 long, long, brutal months of long haulers, I am back to better than I was before. I am a scientist living in Pennsylvania (USA). In August of 2020, I joined a COVID survivors group. There I met Dr. Ade Wentzel from Port Elizabeth (South Africa). Ade and his colleagues, Robert Miller (Cape Town) and Guy Richards (Johannesburg), developed a diet based protocol based on their research COVID-19: NAD+ deficiency may predispose the aged, obese and type2 diabetics to mortality through its effect on SIRT1 activity.

Long haulers syndrome is the depletion of NAD+ (nicotinamide adenine dinucleotide). NAD+ is involved in cell creation, maintenance, metabolism, and regulating cell processes. COVID both increases the breakdown of NAD+ and decreases the production of NAD+. The result is the same as the disease pellagra. A niacin deficiency results in pellagra. Long haulers syndrome is COVID-19 Induced Secondary Pellagra (CISP).

Once I started the protocol, my symptoms diminished and then disappeared completely. In fact three weeks later, my quality of life improved back to when I was 20 years younger. Obviously, I was NAD+ deficient prior to contracting the virus, and COVID depleted my NAD+ even further (bringing me close to death.)

THE PROTOCOL is:
Niacin B3, Vitamin D, Vitamin C, Quercetin, Zinc, and Selenium

Here’s what I’m doing:
Vitamin C, Quercetin and Selenium you can get from your diet. Careful not too much Selenium. I eat 1 Brazil nut a day. The Vitamin D is best gotten from 15 minutes a day in the sun. You can take 1000iu, too. A 15mg zinc/day supplement. The niacin is the trickiest and most important part. You need to make sure you get the right kind. Non-flush is no good. You can start with 35mg/day, but it will depend on your deficiency. I take 50-75mg with each meal. Here’s the walk through on getting the niacin right. It is recommended I stay on the protocol for 6 months. This is how Ade and Rob walk me through getting the correct Niacin B3.

COVID-19 Index

bookmark_borderGreenland Ice Sheet: largest single contributor to rising sea levels.

Dynamic ice loss from the Greenland Ice Sheetdriven by sustained glacier retreat

by Michalea D. King, Ian M. Howat, Salvatore G. Candela, Myoung J. Noh, Seonsgu Jeong, Brice P. Y. Noël, Michiel R. van den Broeke3, Bert Wouters, Adelaide Negrete

Greenland-Ice-Sheet.pdf

The Greenland Ice Sheet is losing mass at accelerated rates in the 21st century, making it thelargest single contributor to rising sea levels. Fasterflow of outlet glaciers has substantiallycontributed to this loss, with the cause of speedup, and potential for future change, uncertain.

“The ice sheet is now in this new dynamic state, where even if we went back to a climate that was more like what we had 20 or 30 years ago, we would still be pretty quickly losing mass,” Ian Howat, co-author of the study and a professor at Ohio State University, said.

CNN reports, “Sea levels are projected to rise by more than 3 feet by the end of the century, wiping away beaches and coastal properties. Coastal states like Florida, and low-lying island nations are particularly vulnerable. Just 3 feet of sea level rise could put large areas of coastline underwater. Forty percent of the US population resides in coastal areas that are vulnerable to sea level rise.”

“There’s a lot of places, like in Florida especially, where one meter alone would cover a lot of existing land areas,” King said. “And that’s exacerbated when you get storms and hurricanes and things like that, that then cause extra surge on top of a higher baseline.”

“We’ve passed the point of no return but there’s obviously more to come,” Howat said. “Rather than being a single tipping point in which we’ve gone from a happy ice sheet to a rapidly collapsing ice sheet, it’s more of a staircase where we’ve fallen off the first step but there’s many more steps to go down into the pit.”

More articles on Flood Insurance, Rising Sea Levels and Global Warming.

Greenland: Slip Sliding Away

bookmark_borderMethane Emissions Soar to Record High

Methane is one of the most dangerous greenhouse gases… and, is part of an exponential negative feedback loop:
Methane in the atmosphere causes the climate to warm
A warmer climate causes permafrost to melt…
Releasing more methane and CO2…
Making the climate warmer…
Melting more permafrost…
Releasing more methane and CO2…
Melting, more permafrost, etc., etc., etc

“And is there a tipping point that could trigger rapid thaw? Scientists estimate that there is about twice as much carbon stored in permafrost as circulating in the atmosphere. … Once the organic matter within permafrost decomposes and releases CO2 and methane, there is no getting it back,” said Dr Christina Schädel.

In a report for Stanford University, Josie Garthwaite states:

lobal emissions of methane have reached the highest levels on record. Increases are being driven primarily by growth of emissions from coal mining, oil and natural gas production, cattle and sheep ranching, and landfills.

Between 2000 and 2017, levels of the potent greenhouse gas barreled up toward pathways that climate models suggest will lead to 3-4 degrees Celsius of warming before the end of this century. This is a dangerous temperature threshold at which scientists warn that natural disasters, including wildfires, droughts and floods, and social disruptions such as famines and mass migrations become almost commonplace. The findings are outlined in two papers published July 14 in Earth System Science Data and Environmental Research Letters by researchers with the Global Carbon Project, an initiative led by Stanford University scientist Rob Jackson.

Globally, fossil fuel sources and cows are twin engines powering methane’s upward climb. “Emissions from cattle and other ruminants are almost as large as those from the fossil fuel industry for methane,” Jackson said. “People joke about burping cows without realizing how big the source really is.”

Throughout the study period, agriculture accounted for roughly two-thirds of all methane emissions related to human activities; fossil fuels contributed most of the remaining third. However, those two sources have contributed in roughly equal measure to the increases seen since the early 2000s.

Methane emissions from agriculture rose to 227 million tons of methane in 2017, up nearly 11 percent from the 2000–2006 average. Methane from fossil fuel production and use reached 108 million tons in 2017, up nearly 15 percent from the earlier period.

Amid the coronavirus pandemic, carbon emissions plummeted as manufacturing and transportation ground to a halt. “There’s no chance that methane emissions dropped as much as carbon dioxide emissions because of the virus,” Jackson said. “We’re still heating our homes and buildings, and agriculture keeps growing.”

The Human Induced Climate Change Experiment

bookmark_borderCOVID-19 Durability, Enhancement, Infection and Exposure?

I asked:
have you seen any studies on COVID immunity durability and enhancement?

No, but here is some material on the difficulty of testing for
COVID infection and COVID exposure

Article by lowe in science blogs:

“from The Lancet on a large study in Spain. Testing tens of thousands of people across the country continues to show that (on average) only about 5% of the population is seropositive (that is, has antibodies to the virus).”

“at least one-third of the people who now test positive never showed any symptoms at all. ”

“we are still not sure if this means that 95% of the Spanish population has never been exposed to the virus, because we don’t know how many people might have cleared it without raising enough of an antibody response to still be detectable. ”

” 40% of asymptomatic patients went completely seronegative during their convalescence.”

“At a minimum, you’d want to know antibody levels over time, T-cell response over time, and (importantly) what a protective profile looks like for both of those. We barely have insight into any of this: the large-scale data are just a snapshot of antibody levels, and that’s not enough.”

“We could have people who look vulnerable but aren’t – perhaps they show no antibodies, but still have a protective T-cell response. Or we could have people who look like they might be protected, but aren’t – perhaps they showed an antibody response many weeks ago that has now declined, and they don’t have protective levels of T-cells to back them up.”

https://blogs.sciencemag.org/pipeline/archives/2020/07/07/more-on-t-cells-antibody-levels-and-our-ignorance

This references a twitter thread by Bleicher:

” Virus must get through mucous membranes, a physical barrier. 2. Innate immunity can destroy virus before it starts. 3. Neutralizing antibodies and memory B cells protect against future infection 4. T cells destroy infected cells”

https://twitter.com/pbleic/status/1278357445023109121

Note that it is much more difficult to test for T-cell and B-cell response than it is to test for antibodies. Very few results have been published for T and B cell response compared to the antibody serological results.

sidd

COVID-19 Index

bookmark_borderThe Real Mortality Rate of COVID-19

By Daniel Brouse, scientist and statistician
July 7, 2020

The Mortality Rate for COVID-19 in the State of Pennyslvania is 13.68%.

I grew weary of people trying to argue the mortality rate in the state of PA. I was being conservative and lazy… estimating it around 7.5% (deaths / cases = mortality rate). The science deniers were still telling me it is no worse than the flu. So, this is what I had to do.

I contacted the State of Pennsylvania and asked:
Can you tell me how PA is counting “cases” — 91,299 cases. Technically, traditionally… the mortality rate = deaths / cases.
A case: symptomatic individual that presents themselves to a healthcare facility/hospital. I can not find a definition of a “COVID case” on the PA website. It looks like we are counting all the asymptomatic people that are being tested? That is not how the mortality rate for the flu, nor any other disease, is calculated. Normally, with the flu and all other diseases, the infected are counted as “symptomatic presenting as a case in a healthcare facility.” It is a doctor’s “case”. That is not how the COVID mortality rate is being calculated. This is important because, if we deviate toward counting others as “infected”, we can never get an accurate accounting. We can never know the number of asymptomatic infected. That is why only symptomatic patients are, or should be, counted.

The State replied: “Positive viral lab tests = confirmed cases. They have also included 634 patients in the count who have a positive serology test and either COVID-19 symptoms or a high-risk exposure.”

So, to calculate an accurate mortality rate to compare to a mortality rate for the flu, I need to subtract out how many of the positive tests were from asymptomatic individuals, and then I need to estimate the time from “testing positive” to death.

The second part is easier. Average time from testing to death for most deaths is about 45 days, but some are hospitalized for 100 days after being symptomatic for days (like Nick Cordero at age 41). In an effort to pacify the conservatives, I’ll go with around 45 days.
45 days ago there was a total of 62,000 people testing positive.
Now, I’ll be even more conservative and say 80% of those tested were symptomatic.
62,000 x .8 = 49,600
The reported deaths as of today is 6,787.

6,787 deaths / 49,600 cases = 13.68% mortality rate for COVID-19 in the state of PA on 7/7/2020

For the most recent flu season there were 102 deaths out of 129,912 lab confirmed cases. (https://www.health.pa.gov/topics/disease/Flu/Pages/2019-20-Flu.aspx)

102 deaths / 129,912 cases = .079% mortality rate for the flu in the state of PA on 7/7/2020

MORTALITY RATES IN THE STATE OF PENNSYLVANIA
COVID-19 = 13.68%
Flu = .079%

More Than 900,000 People Have Died Of COVID-19 In U.S (May 6, 2021 / IHME)

More Science Based Information on the COVID Mortality Rate

COVID-19 Index

bookmark_borderThe Economic Costs of COVID Re-openings

The mortality rate for COIVD is between 7-8% in PA. (* The actual mortality rate is closer to 14%) It has been since the pandemic started, and still is. An estimated additional 15% of the recovered have permanent disabilities. What is the cost of life and health on the economy? The benefit-cost analysis of a human life is generally accepted as US$10 million/dead person. [It is also referred to as the cost of life, value of preventing a fatality (VPF) and implied cost of averting a fatality (ICAF).]

But, what is the cost of lifelong disability? (COD/cost of a disability) That cost is much more difficult to calculate. How much does it cost to keep me alive? How much pain and suffering must I endure? And, how widely does the cost vary from disability to disability? If I need O2 the rest of my life… or if my leg is amputated… or neurological damage, etc.?

So, if the economy benefits by X amount by reopening early… what is the offsetting cost (X-VPF-COD)?
It seems to me this is a no-brainer. Without a doubt, it will be a negative number: X < VPF+COD Answer: Stop the spread. Stop the dead. Make the bread.

COVID-19 Index

bookmark_borderFar More Properties At Risk: Flood Insurance Exposure

The last link is a map that lets you find flood risk exposed properties by zipcode

Far more properties at flood risk than estimated:

” far more U.S. homes — an estimated 14.6 million — face flooding conditions on par with those in FEMA’s 100-year floodplain. In contrast, FEMA’s maps classify 8.7 million homes as carrying substantial risk.”

“The true figure climbs to 16.2 million homes by 2050 when the effects of climate change are included”

“21 states have no requirements for sellers to disclose flood risk”

https://www.politico.com/news/2020/06/29/study-flood-risks-federal-estimates-344442

“Everyone is exposed ”

Fannie:”exposure to flooding-related risk may not be fully captured under NFIP insurance coverage”

Freddie: “Some of the varied impacts of climate change — rising sea levels, changing rainfall and flooding patterns, increasing temperatures — may not be insurable”

NBER: “homes in flood plains are overvalued by $34 billion”

“Homebuilding and mortgage originations in floodplains have risen steadily over the years even as the number of flood insurance policies has fallen”

” where the risk of flooding is highest, the number of policies held has shrunk even faster, according to FEMA. In 2008, there were 2.5 million residential structures in floodplains insured by the NFIP. By the end of 2019, that number had fallen to fewer than 1.8 million.”

“While Fannie and Freddie don’t screen loans for disaster risk, the mortgage lenders who sell them loans do, a dynamic that could be pushing even more risky loans onto the companies’ books.”

https://www.politico.com/news/2020/06/08/borrowed-time-climate-changemortgage-market-304130
https://prod.nber.org/papers/w26807
https://www.politico.com/f/?id=00000172-86b3-d4f1-adf3-87b7f1300000

https://www.vice.com/en_us/article/9353dv/this-map-shows-which-us-homes-will-flood-over-the-next-30-years-due-to-climate-change

https://floodfactor.com

More articles on Flood Insurance, Rising Sea Levels and Global Warming.

bookmark_borderClimate Change and Mortgage Change

New York Times
By Christopher Flavelle
June 19, 2020

Rising Seas Threaten an American Institution: The 30-Year Mortgage

Climate change is starting to transform the classic home loan, a fixture of the American experience and financial system that dates back generations.

WASHINGTON — Up and down the coastline, rising seas and climate change are transforming a fixture of American homeownership that dates back generations: the classic 30-year mortgage.

Home buyers are increasingly using mortgages that make it easier for them to stop making their monthly payments and walk away from the loan if the home floods or becomes unsellable or unlivable. More banks are getting buyers in coastal areas to make bigger down payments — often as much as 40 percent of the purchase price, up from the traditional 20 percent — a sign that lenders have awakened to climate dangers and want to put less of their own money at risk.

And in one of the clearest signs that banks are worried about global warming, they are increasingly getting these mortgages off their own books by selling them to government-backed buyers like Fannie Mae, where taxpayers would be on the hook financially if any of the loans fail.

“Conventional mortgages have survived many financial crises, but they may not survive the climate crisis,” said Jesse Keenan, an associate professor at Tulane University. “This trend also reflects a systematic financial risk for banks and the U.S. taxpayers who ultimately foot the bill.”

The trends foreshadow a broader reckoning. The question that matters, according to researchers, isn’t whether the effects of climate change will start to ripple through the housing market. Rather, it’s how fast those effects will occur and what they will look like.

The change has already begun. It’s not only along the nation’s rivers and coasts where climate-induced risk has started to push down home prices. In parts of the West, the growing danger of wildfires is already making it harder for homeowners to get insurance.

But the threat that climate change poses to the 30-year mortgage is different, striking at an American social institution that dates from the Great Depression. Before that, many home loans required owners to pay lenders back just a few years after buying a house, which led to waves of defaults and homelessness, according to Andrew Caplin, a professor of economics at New York University.

In response, the federal government created the Federal Housing Administration, which in turn standardized the way Americans finance their homes.

There was nothing magical about a period of 30 years, Dr. Caplin said; it simply proved useful, making payments predictable and affordable by stretching them out over decades. “It was designed from a viewpoint of a consumer, who wouldn’t find it too hard to understand exactly what they had committed to,” Dr. Caplin said.

But now, as the world warms, that long-term nature of conventional mortgages might not be as desirable as it once was, as rising seas and worsening storms threaten to make some land uninhabitable. A retreat from the 30-year mortgage could also put homeownership out of reach for more Americans.

Changes to the housing market are just one of myriad ways global warming is disrupting American life, including spreading disease and threatening the food supply. It could also be one of the most economically significant. During the 2008 financial crisis, a decline in home values helped cripple the financial system and pushed almost nine million Americans out of work.

But increased flooding nationwide could have more far-reaching consequences on financial housing markets. In 2016, Freddie Mac’s chief economist at the time, Sean Becketti, warned that losses from flooding both inland and along the coasts are “likely to be greater in total than those experienced in the housing crisis and the Great Recession.”

If climate change makes coastal homes uninsurable, Dr. Becketti wrote, their value could fall to nothing, and unlike the 2008 financial crisis, “homeowners will have no expectation that the values of their homes will ever recover.”

In 30 years from now, if global-warming emissions follow their current trajectory, almost half a million existing homes will be on land that floods at least once a year, according to data from Climate Central, a research organization. Those homes are valued at $241 billion.

Currently, new research shows banks rapidly shifting mortgages with flood risk off their books and over to organizations like Fannie Mae and Freddie Mac, government-sponsored entities whose debts are backed by taxpayers. In a paper this month in the journal Climactic Change, Dr. Keenan and Jacob T. Bradt, a doctoral student at Harvard University, described the activity, which suggests growing awareness among banks that climate change could cause defaults.

Tellingly, the lenders selling off coastal mortgages the fastest are smaller local banks, which are more likely than large national banks to know which neighborhoods face the greatest climate risk. “They have their ears to the ground,” Dr. Keenan said.

In 2009, local banks sold off 43 percent of their mortgages in vulnerable zones, Dr. Keenan and Mr. Bradt found, about the same share as other areas. But by 2017, the share had jumped by one-third, to 57 percent, despite staying flat in less vulnerable neighborhoods.

If coastal mortgages defaulted on those loans, it could cause losses for Fannie and Freddie. That pain could spread to taxpayers: In 2008, the two firms required $187 billion in public aid to stay solvent. They later repaid the money.

In a separate working paper with Marco Tedesco and Carolynne Hultquist of Columbia University’s Lamont-Doherty Earth Observatory, Dr. Keenan found banks protecting themselves in other ways, such as lending less money to home buyers in vulnerable areas, relative to the value of the homes.

Typically, a bank will lend about 80 percent of the cost of a house, with the buyer putting down the other 20 percent. But examining several counties particularly exposed to rising seas, the researchers found that a growing share of mortgages had required down payments between 21 percent and 40 percent — what Dr. Keenan called nonconventional loans.

In coastal Carteret County, N.C., the share of nonconventional mortgages increased by 14 percent between 2006 and 2017 in the areas most exposed to sea-level rise. That change can’t be explained by the general trend there: In the rest of Carteret County, nonconventional loans became less common during the same period.

Similarly in St. Johns County, Fla., south of Jacksonville, between 2006 and 2017, the share of nonconventional loans in the most vulnerable areas increased by 6 percent, while falling 22 percent in the rest of the county. “You’re seeing a statistically significant trend,” Dr. Keenan said.

The Mortgage Bankers Association, a trade group, declined to comment directly on the findings. Pete Mills, the association’s senior vice president of residential policy, cited the requirement for homeowners to buy insurance.

“Lenders make sure all properties are properly insured,” Mr. Mills said in a statement. “For loans in Special Flood Hazard Areas, flood insurance is required,” he added, referring to areas the Federal Emergency Management Agency has determined have a high flood risk.

Fannie Mae and Freddie Mac said, “Any loan located in FEMA-designated Special Flood Hazard Areas must have flood insurance in order for the loan to be purchased by Freddie Mac or Fannie Mae.”

But flood insurance isn’t likely to address the problem, Dr. Keenan said, because it doesn’t protect against the risk of a house losing value and ultimately becoming unsellable.

Lenders aren’t the only ones who seem to be inching away from traditional 30-year mortgages in risky areas. More homeowners are also taking out a type of mortgage that is less financially painful for a borrower to walk away from if a home becomes uninhabitable because of rising seas. These are known as interest-only mortgages — the monthly payment covers only the interest on the loan, and doesn’t reduce the principal owed.

Under normal circumstances, this kind of loan sounds like a bad deal: It’s a loan you can never pay off with the regular monthly payments. However, it also means buyers aren’t sinking any more of their own money into the property beyond a down payment. That’s an advantage if you think the property may become unlivable.

“A household that expects the house will be flooded within a decade, say, is unlikely to value the accumulation of equity in this house,” said Amine Ouazad, an associate professor of real estate economics at HEC Montreal who has researched the practice. “The ability to walk away from a mortgage in case of major floods or sea-level rise is a feature.”

In new research this month, Dr. Ouazad found that, since the housing crash, the share of homes with fixed-rate, 30-year mortgages has declined sharply — to less than 80 percent, as of 2016 — in areas most exposed to storm surges. In the rest of the country, the rate has stayed constant, at about 90 percent of home loans.

Part of the difference was the interest-only loans, Dr. Ouazad found. More than 10 percent of homeowners in those areas had interest-only loans in 2016, compared with just 2.3 percent in other ZIP Codes. The work hasn’t been peer-reviewed, and more research is needed, Dr. Ouazad said. But he said there’s reason to think climate risks are part of the explanation.

The tougher question, according Carolyn Kousky, executive director of the Wharton Risk Center at the University of Pennsylvania, is what happens after that, when people quite simply no longer want to live in homes that keep flooding. “What happens when the water starts lapping at these properties, and they get abandoned?” she said.

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